Dave, N.*, Judd, J.*, Decker, A., Winslow, W., Sarette, P., Espinosa, O., Tallino, S., Bartholomew, S., Bilal, A., Sandler, J., McDonough, I., Winstone, J., Blackwood, E., Glembotski, C., Karr, T., Velazquez, R. (2023). Dietary choline intake is necessary to prevent systems-wide organ pathology and reduce Alzheimer’s disease hallmarks. Aging Cell 22(2):e13775. (*Co-Authors)
Tallino, S., Winslow, W., Velazquez, R. (2023). Trajectory of amyloid pathology in a Down syndrome model. Aging 15(2):295-297.
Tallino, S., Winslow, W., Bartholomew, S., Velazquez, R. (2022). Temporal and brain region specific elevations of soluble Amyloid Beta40-42 in the Ts65Dn mouse model of Down syndrome and Alzheimer's disease. Aging Cell e13590.
Winslow, W., Mcdonough, I., Tallino, S., Decker, A., Vural, A., Velazquez, R. (2021). IntelliCage automated behavioral phenotyping reveals behavior deficits in the 3xTg-AD mouse model of Alzheimer's disease associated with brain weight. Frontiers in Aging Neuroscience 13:720214.
Mifflin, M., Winslow, W., Surendra, L., Tallino, S., Vural, A., and Velazquez, R. (2021). Sex differences in the IntelliCage and the Morris water maze in the APP/PS1 mouse model of amyloidosis. Neurobiology of Aging 101:130-140.
Morrell, A., Tallino, S., Yu, L., and Burkhead, J. L. (2017). The role of insufficient copper in lipid synthesis and fatty-liver disease. IUBMB Life 69(4):263-270.
Tallino, S., Duffy, M., Ralle, M., Cortés, M. P., Latorre, M., and Burkhead, J. L. (2015). Nutrigenomics analysis reveals that copper deficiency and dietary sucrose up-regulate inflammation, fibrosis, and lipogenic pathways in a mature rat model of nonalcoholic fatty liver disease. Journal of Nutritional Biochemistry 26(10):996-1006.
Tallino, S. (2014). Copper deficiency and sucrose consumption: Factors in the “western diet” and their relationship to non-alcoholic fatty liver disease and cognitive decline (Order No. 1563531). Available from ProQuest Dissertations and Theses Global. (1612508555).